The Little Black Book of Cardiology (Jones And Bartlett's Little Black Book Series) [John A. Sutherland] on ruthenpress.info *FREE* shipping on qualifying offers. The Little Black Book of Cardiology (Second Edition) on ruthenpress.info *FREE* shipping on qualifying offers. Quick reference to Important Info. Small coffee stain . The Little Black Book of Cardiology, Second edition is a pocket-sized handbook with comprehensive, concise, evidence-based information on diagnosing and.
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The Little Black Book Of Cardiology by John A. Sutherland, , available at Book Depository with free delivery worldwide. The Little Black Book of Cardiology is a convenient resource offering quick access to vital information and makes a great reference for solving pressing problems. The Little Black Book of Cardiology book. Read reviews from world's largest community for readers. Thoroughly revised and updated, the second edition of.
There is no other way to practice heart medicine. For even if the heart is not the seat of the emotions, it is highly responsive to them.
In the late s chest compressions were discovered to help raise blood pressure and now are common practice in resuscitations. In advanced open heart surgery was extremely rare being conducted by only one doctor using cross-circulation another healthy person as a donor.
The doctor came to the United States in to continue his research. The automatic defibrillator was approved by the FDA in Even though there has been a drop in cardiovascular mortality, we still must continue on the path of research and discovery. Pimple Popper!
In modern day cardiology and cardiac surgery, the electrocardiogram ECG receives only a cursory glance from those who are not electrophysiologists so it was a surprise to find such an excellent chapter on the subject, including guidance on how to interpret an exercise ECG.
A chapter on echocardiography is up-to-date with details of semi-quantitative assessment of valvular regurgitation, together with highly practical points such as the distinction between a pericardial and a pleural effusion.
Cardiac physiology is well reviewed in an early chapter and appears again within the cardiac catheterisation chapter where measurement of cardiac output and shunt calculations are clearly explained.
There are three chapters, which deal with cardiopulmonary bypass and its complications. Here again, there are very clear diagrams which explain well cannulation and venting.
In the sections on specific groups of diseases there is a strong reliance on evidence base and guidelines.
It was therefore a little surprising to find no mention of informed consent or the details of risk assessment. There seem to be very few errors but I was struck by the bald statement that bicuspid aortic valve has an autosomal dominant inheritance pattern.
There are an excellent set of questions relating to important trials which are very contemporary. Overall, this is an exceptionally well-produced book which is a delight to read and is likely to run to several more editions. For instance, research by Armando Meyer and colleagues published in the February EHP found that the insecticide chlorpyrifos affects cell signaling cascades critical to cardiac homeostasis.
Problems may be caused not just by a chemical, but by its metabolites. In a study reported in the September issue of Toxicological Sciences, Dunnick and NIEHS colleague Abraham Nyska found that bis 2-chloroethoxy methane caused mitochondrial damage in hearts in a rodent model system.
They hypothesized that the thiodiglycolic acid, a metabolite of bis 2-chloroethoxy methane as well as many other chemicals, causes this chemical-related mitochondrial damage and heart toxicity. Dunnick and Nyska observed a biphasic response: initial damage to myocytes was repaired in an apparent temporary adaptive response that the animals were no longer able to launch as they aged. Other areas of concern include genetic variations and expression, gene polymorphisms, oxidative stress, protein expression, and post-translational modifications.
In addition, many researchers suspect indirect links with the immune, pulmonary, and neurological systems. Seeking the Heart of the Matter Millions of dollars are being pumped into environmental cardiology research. The year study will evaluate about 8, people in six states, representing a variety of ethnic groups, for clinical and subclinical effects such as heart attack, stroke, and atherosclerosis. The study, expected to conclude in , will investigate a range of CVD effects, in part through intensive personal monitoring, and will address seasonal and geographic variations.
The topics range from acute effects of particulates on the autonomic nervous system to chronic effects of particulates on atherosclerosis, Mastin says. A University of Rochester study looking at the effects of particulates on CVD is expected to be published by November Dockery is looking at whether particulate exposures may make internal cardiac defibrillators fire more frequently. Many other related studies are being conducted around the world. Mastin, whose branch administers this program, says research funded under these grants is examining how prenatal exposures influence the risk of cardiovascular birth defects.
The fledgling program, begun in , is designed to eventually provide extensive data documenting links between the presence of environmental agents, exposures, and ensuing diseases, including CVD. Although the links between chemical exposures and CVD are becoming more widely recognized, few public health agencies have responded yet.
NHLBI, too, acknowledges the issue of chemical exposures, says George Sopko, a cardiologist at that institute, but has not given it much weight yet. Few, if any, state health programs address environmental cardiology in any significant way, according to several state health officials. Riding the Learning Curve The venues for professionals interested in learning more about the links between environmental agents and CVD are expanding.
One journal that focuses extensively on related issues of cardiovascular toxicities of drugs, novel therapies, and environmental pollutants is Cardiovascular Toxicology, which began publishing in Interested doctors may soon be able to learn more through continuing medical education programs. The EPA is working on a certification program to educate doctors about ozone and respiratory effects, says agency environmental health scientist Susan Stone, and the agency anticipates following that up with a program on particulates that will cover cardiovascular effects.
At the moment, though, doctors have no accepted medical treatments that are known to be effective in reducing the effects of chemicals on CVD, says Robert D. Brook, who is lead author of the AHA Scientific Statement, an assistant professor of medicine at the University of Michigan, and a practicing physician.
Instead, concerned people should avoid exposures to the extent possible, he says. Daily reports on local particulate and ozone levels can help people decide whether to limit activity and thus, exposure. It devotes about half its space to the effects of a few common pollutants on the cardiovascular system.
The other half addresses respiratory effects. In the agency released an educational brochure on particulates that folded in some information on cardiovascular effects.
Reaching a Regulatory Threshold Given the relatively early stages of wide-scale research into the links between chemicals and CVD, it likely will be some time before any regulations change.
Greg Dana, vice president of environmental affairs with the Alliance of Automobile Manufacturers, hopes she is correct. He says that vehicle pollution, which accounts for a large part of the environmental load of particulates and other air pollutants, has already been addressed enough. Both of these rules also contain provisions to remove sulfur from gasoline and diesel fuel. If additional regulations are adopted, they might address concentrations of allowable exposure, time period of exposure, and vulnerable populations, says Farland.